Scopus İndeksli Yayınlar Koleksiyonu / Scopus Indexed Publications Collection
Permanent URI for this collectionhttps://hdl.handle.net/11147/7148
Browse
3 results
Search Results
Correction Automating Software Size Measurement From Python Code Using Language Models (Vol 33, 19, 2026)(Springer, 2025) Tenekeci, Samet; Unlu, Huseyin; Gul, Bedir Arda; Keles, Damla; Kucuk, Murat; Demirors, OnurArticle Automating Software Size Measurement from Python Code Using Language Models(Springer, 2025) Tenekeci, Samet; Unlu, Huseyin; Gul, Bedir Arda; Keles, Damla; Kuuk, Murat; Demirors, OnurSoftware size is a key input for project planning, effort estimation, and productivity analysis. While pre-trained language models have shown promise in deriving functional size from natural-language requirements, measuring size directly from source code remains under-explored. Yet, code-based size measurement is critical in modern workflows where requirement documents are often incomplete or unavailable, especially in Agile development environments. This exploratory study investigates the use of CodeBERT, a pre-trained bimodal transformer model, for measuring software size directly from Python source code according to two measurement methods: COSMIC Function Points and MicroM. We construct two curated datasets from the Python subset of the CodeSearchNet corpus, and manually annotate each function with its corresponding size. Our experimental results show that CodeBERT can successfully measure COSMIC data movements with up to 91.4% accuracy and generalize to the functional, architectural, and algorithmic event types defined in MicroM, reaching up to 81.5% accuracy. These findings highlight the potential of code-based language models for automated functional size measurement when requirement artifacts are absent or unreliable.Article Citation - WoS: 4Citation - Scopus: 4Integrative Biological Network Analysis To Identify Shared Genes in Metabolic Disorders(Institute of Electrical and Electronics Engineers, 2022) Tenekeci, Samet; Işık, ZerrinIdentification of common molecular mechanisms in interrelated diseases is essential for better prognoses and targeted therapies. However, complexity of metabolic pathways makes it difficult to discover common disease genes underlying metabolic disorders; and it requires more sophisticated bioinformatics models that combine different types of biological data and computational methods. Accordingly, we built an integrative network analysis model to identify shared disease genes in metabolic syndrome (MS), type 2 diabetes (T2D), and coronary artery disease (CAD). We constructed weighted gene co-expression networks by combining gene expression, protein-protein interaction, and gene ontology data from multiple sources. For 90 different configurations of disease networks, we detected the significant modules by using MCL, SPICi, and Linkcomm graph clustering algorithms. We also performed a comparative evaluation on disease modules to determine the best method providing the highest biological validity. By overlapping the disease modules, we identified 22 shared genes for MS-CAD and T2D-CAD. Moreover, 19 out of these genes were directly or indirectly associated with relevant diseases in the previous medical studies. This study does not only demonstrate the performance of different biological data sources and computational methods in disease-gene discovery, but also offers potential insights into common genetic mechanisms of the metabolic disorders.