Imatinib Induces Autophagy Through Increasing the Expression Levels of Beclin-1 and Atg-5 Genes in Both Parental Sensitive and Imatinib-Resistant K562 Cells

dc.contributor.author Baran, Yusuf
dc.contributor.author Baran, Yusuf
dc.contributor.author Can, Geylani
dc.contributor.author Ekiz, Hüseyin Atakan
dc.contributor.author Ekiz, Hüseyin Atakan
dc.contributor.author Genç, S. G.
dc.contributor.other 04.03. Department of Molecular Biology and Genetics
dc.contributor.other 04. Faculty of Science
dc.contributor.other 01. Izmir Institute of Technology
dc.date.accessioned 2021-01-24T18:32:01Z
dc.date.available 2021-01-24T18:32:01Z
dc.date.issued 2009
dc.description 14th Annual Meeting of the European-Hematology-Association en_US
dc.description.abstract [No abstract available] en_US
dc.description.sponsorship European Hematol Assoc en_US
dc.identifier.issn 0390-6078
dc.identifier.issn 1592-8721
dc.identifier.uri https://hdl.handle.net/11147/10031
dc.language.iso en en_US
dc.publisher Ferrata Storti Foundation en_US
dc.relation.ispartof Haematologica en_US
dc.rights info:eu-repo/semantics/openAccess en_US
dc.title Imatinib Induces Autophagy Through Increasing the Expression Levels of Beclin-1 and Atg-5 Genes in Both Parental Sensitive and Imatinib-Resistant K562 Cells en_US
dc.type Conference Object en_US
dspace.entity.type Publication
gdc.author.institutional Baran, Yusuf
gdc.author.institutional Can, Geylani
gdc.author.institutional Ekiz, Hüseyin Atakan
gdc.coar.access open access
gdc.coar.type text::conference output
gdc.description.department İzmir Institute of Technology. Molecular Biology and Genetics en_US
gdc.description.departmenttemp [Baran, Y.; Can, G.; Ekiz, H. A.] Izmir Inst Technol, Izmir, Turkey; [Genc, S. G.] Dokuz Eylul Univ, Dept Oncol, Izmir, Turkey en_US
gdc.description.endpage 233 en_US
gdc.description.publicationcategory Makale - Uluslararası Hakemli Dergi - Kurum Öğretim Elemanı en_US
gdc.description.scopusquality Q1
gdc.description.startpage 233 en_US
gdc.description.volume 94 en_US
gdc.description.wosquality Q1
gdc.identifier.wos WOS:000266931900571
gdc.index.type WoS
gdc.wos.citedcount 0
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